Atherosclerosis is a general term given to many diseases which result in a hardening and thickening of the walls of the circulatory system. It starts when monocytes (white blood cells) input the walls of their arteries in certain locations and are changed into cells that bring and accumulate fatty deposits. These areas become thickened and difficult and are referred to atheromas.
Decreased blood flow and thrombosis become probably as the disorder progresses. The long-term consequences are grave and include cardiovascular disease and stroke. Tasigna Atherosclerosis and its effects lead to it being responsible for more deaths than all other factors combined. In its earlier phases, the development of atherosclerosis could be stopped and even reversed with the correct lifestyle changes.
There's continuing research into the mechanics supporting the build-up of plaque. Recent studies have linked this to an inflammation reaction. This new understanding sheds light on why it's that elevated cholesterol levels aren't necessarily the principal indicator of cardiovascular disease from atherosclerosis. Not many individuals with high cholesterol wind up with atherosclerosis, and not many individuals with atherosclerosis have elevated cholesterol.
The low-grade disease now appears to underlie what has been formerly regarded as a very simple build-up of plaque.
Particular risk factors influence the discharge of compounds involved in the inflammatory procedure. These contribute not just to the creation of plaque however into the formation of blood clots. Atherosclerosis is thought to involve compounds involved in the inflammatory reaction and cells which are characteristic of inflammation.
Research also indicates that an illness can cause or even cause atherosclerosis. Chlamydia has been proven to have a substantial association with the plaque.